attention deficit disorder :: essays research papers

In 1917 and 1918 there was a worldwide outbreak of encephalitis. numerous children showed ADD like symptoms subsequentlywards, leading doctors to further speculate that the disorder was the get out of brain damage, resulting in perhaps the first names for ADD, Post-Encephalitic Disorder and nominal Brain Damage. When it was later learned that these children were not brain damaged after all, the name changed to Minimal Brain Dysfunction. In 1937 doctors first reported leaven that stimulant medication helped relieve the symptoms of active children. These reports were largely ignored until the 1950s, when there was a rapid increase in the use of drug therapy in psychiatry. By the mid 1960s, stimulant medication had become a frequent treatment for hyperactivity. In 1960 Stella Chase and other researchers began to write about "hyperactive child syndrome," which they believed to have a biological, rather than environmental cause. In 1965 the American Psychiatric Associatio n established a diagnostic category for what was instantaneously called Hyperkinetic Reaction of Childhood. In spite of the work of Chase and others, this condition was becalm largely believed to be caused by the childs environment, rather than being biological in origin. As a result, poor parenting was often blamed for ADD behavior. all over the next 15 years, research began to reveal the neurological origin of ADD. one and only(a) of the many who studied ADD in the 1970s was Canadian psychologist Virginia Douglas. She is credited with identifying quatern major characteristics of the syndrome deficits in attention and effort, impulsivity, problems in regulating arousal levels and a need for immediate reinforcement. It was largely due to Douglass work that in 1980 the American Psychiatric Association established a new diagnostic category, charge Deficit Disorder With or Without Hyperactivity. In 1987 the APA revised the name to heed Deficit-Hyperactivity Disorder. More recent ly, with growing